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The Science Behind how your Children are affected

Mounting evidence points to a clear biological cascade, moving EHS from a "medically unexplained" condition to one with identifiable pathophysiological mechanisms.

The Pathophysiological Cascade: How EMFs Impact the Body

Man-Made EMF Exposure

(Pulsed, Polarized)

 

VGCC Activation

(Voltage-Gated Calcium Channels)

 

Oxidative Stress

(Increased ROS Production)

 

Low-Grade Neuroinflammation

Activation of microglia, astrocytes, and mast cells in the central nervous system.

Blood-Brain Barrier (BBB) Disruption

Compromised barrier allows inflammatory cells to enter the brain.

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Genetic Predisposition

Specific variations in detoxification pathway genes (e.g., GSTT1, GSTM1) can increase EHS risk by nearly 10-fold.

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Inflammatory Markers

Elevated levels of Histamine (2-10x higher) and Heat Shock Proteins (HSP) indicate chronic, low-grade inflammation.

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BBB Disruption Markers

Increased Protein S100B and Nitrotyrosine in the blood confirm a compromised blood-brain barrier.

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Altered Melatonin

A notable decrease in the excretion of 6-hydroxymelatonin sulfate is found in ~90% of EHS cases, impacting sleep and antioxidant defense.

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Autoimmunity

Anti-myelin autoantibodies are detected in ~15% of cases, suggesting the immune system may attack nerve sheathings.

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Heart Rate Variability

EMF exposure can alter HRV, indicating a physiological stress response and a disturbed circadian rhythm.

Electrohypersensitivity (EHS): An Evidence-Based Infographic

The Path Forward: A Call for Action

Addressing your children's health requires a paradigm shift. We need to update usage policies based on non-thermal biological effects, formal recognition of EHS as a disability, the creation of low-EMF "white zones," and industry-independent research into safer technologies.

Applying the Precautionary Principle and ALARA (As Low As Reasonably Achievable) is essential for a sustainable coexistence with modern technology.

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